We are the combination of four hospitals: the General Hospital, the Children’s Hospital, the Women’s Hospital and the Traumatology, Rehabilitation and Burns Hospital. We are part of the Vall d’Hebron Barcelona Hospital Campus: a world-leading health park where healthcare plays a crucial role.
Below we will list the departments and units that form part of Vall d’Hebron Hospital and the main diseases that we treat. We will also make recommendations based on advice backed up by scientific evidence that has been shown to be effective in guaranteeing well-being and quality of life.
We will guide you from your first visit to the centre, allowing you to find all the departments and make the most of our facilities. Whatever the reason for your visit, we will explain how to get about the hospital.
Prof. Mikko Seppänen, Chief Physician, Specialist in Internal Medicine and Infectious Diseases, Adjunct Professor HUS Helsinki University Hospital, Children and Adolescents, Rare Disease Center- and Inflammation Center, Adult Immunodeficiency Unit- Majakka, Helsinki
"Finnish PIDDs- unexpected autoinflammation in unexpected places"
Finns, a genetically-isolated population do to historical bottleneck events, carry relatively little genomic variation and enrichment of autosomal recessive “Finnish Disease Heritage” (FDH) genes. Some FDH PIDDs display features of inflammasome-mediated autoinflammation, potentially offering new therapeutic targets. Within CVID phenotype - also enriched in Finns - AD NFKB1 mutations cause autoinflammatory features that suggest long sought immunopathogenetic mechanisms of more common inflammatory diseases. A novel noncanonical inflammasomopathy “C/EBPε-associated autoinflammation and immune impairment of neutrophils” (CAIN) raises further interesting questionsFinns, a genetically-isolated population do to historical bottleneck events, carry relatively little genomic variation and enrichment of autosomal recessive “Finnish Disease Heritage” (FDH) genes. Some FDH PIDDs display features of inflammasome-mediated autoinflammation, potentially offering new therapeutic targets. Within CVID phenotype - also enriched in Finns - AD NFKB1 mutations cause autoinflammatory features that suggest long sought immunopathogenetic mechanisms of more common inflammatory diseases. A novel noncanonical inflammasomopathy “C/EBPε-associated autoinflammation and immune impairment of neutrophils” (CAIN) raises further interesting questions.
Host: Infection in Immunocompromised Pediatric Patients psoler@vhebron.net
Cartell
Select the newsletter you want to receive:
By accepting these conditions, you are agreeing to the processing of your personal data for the provision of the services requested through this portal, and, if necessary, for any procedures required by the administrations or public bodies involved in this processing, and their subsequent inclusion in the aforementioned automated file. You may exercise your rights to access, rectification, cancellation or opposition by writing to web@vhebron.net, clearly stating the subject as "Exercising of Data Protection Rights". Operated by: Hospital Universitari Vall d'Hebron - Institut Català de la Salut. Purpose: Manage the user’s contact information. Legitimisation: Express acceptance of the privacy policy. Rights: To access, rectify, and delete personal information data, as well to the portability thereof and to limit and/or oppose their use. Source: The interested party themselves.